Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the flip side, our power to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by amplified pFAK localization in https://pro33login99998.blogerus.com/54693002/what-does-pro33-mean